of histopathologic and functional outcome[J].J Neurotrauma,1999,16(4):299- 309.
[8]Djebaili M,Guo Q,Pettus EH,et al.The neurosteroids progesterone and allopregnanolone reduce cell death,glisis,and functional deficits after traumatic brain injury in rats[J].J Neurotrauma,2005,22(5):106-118.
[9]Kalkhoven E,Wissink S,van der Saag PT,et al.Negative interaction between the RelA(P65) subunit of NFkappaB and the progesterone receptor[J].J Biol Chem,1996,271(13):6217-6224.
[10]MorgantiKossmann MC,Rancan M,Otto VI,et al.Role of cerebral inflammation after traumatic brain injury:a revisited concept[J].Shock,2001,16(4):165-177.
[11]Ghosh S,Karin M.Missing pieces in the NFkappaB puzzle[J].Cell，2002，109(17):81-96.
[12]Shear DA,Galani R,Hoffman SW,et al.Progesterone protects against necrotic damage and behavioral,abnormalities caused by traumatic brain injury[J].Exp Neurol,2002,178(16):59-67.
[13]Stein DG,Hoffman SW.Estrogen and progesterone as neuroprotective agents in the treatment of acute brain injuries[J].Pediat Rehabil,2003,6(2):13-22.
上一页 [1] [2] [3] [4] [5]
您现在的位置： 绿色健康网 >> 医学论文 >> 外科论文 >> 正文