ro T, et al. Serum paraoxonase activity decreases in rheumatoid arthritis [J]. Life Sci, 2003,72(25):2877-2885.
[7] Selek S, Cosar N, Kocyigit A, et al. PON1 activity and total oxidant status in patients with active pulmonary tuberculosis[J]. Clin Biochem, 2008,41(3):140-144.
[8] Yudoh K, Nguyen T, Nakamura H, et al. Potential involvement of oxidative stress in cartilage senescence and development of osteoarthritis: oxidative stress induces chondrocyte telomere instability and downregulation of chondrocyte function[J]. Arthritis Res Ther, 2005,7(2):R380-391.
[9] Draganov D, Stetson P, Watson C, et al. Rabbit serum paraoxonase3 (PON3) is a high density lipoprotein-associated lactonase and protects low density lipoprotein against oxidation [J]. J Biol Chem, 2000, 75(43):33435-33442.
[10] Kobayashi H. Endogenous anti-inflammatory substances, inter-alpha-inhibitor and bikunin [J]. Biol Chem, 2006,387(12):1545-1549.Xu N, Dahlb?覿ck B. A novel human apolipoprotein (apoM) [J]. J Biol Chem, 1999,274(44):31286-1290.
[11] Luo G, Zhang X, Nilsson-Ehle, et al. Apolipoprotein M [J/OL]. Lipids in Health and Disease, 2004:3:21[2004-10-04.]. http://www.lipidworld.com/content/3/1/21
[12] Huang XS, Zhao SP, Hu M, et al. Apolipoprotein M likely extends its anti-atherogenesis via anti-inflammation [J]. Med Hypotheses, 2007,69(1):136-140.
[13] Feingold KR, Shigenaga JK, Chui LG, et al. Infection and inflammation decrease apolipoprotein M expression [J]. Atherosclerosis, 2008,199(1):19-26.
[14] Samuels J, Ng YS, Coupillaud C, et al. Impaired early B cell tolerance in patients with rheumatoid arthritis [J]. J Exp Med, 2005,201(10):1659-1667.
[15] Da RR, Qin Y, Baeten D, et al. B cell clonal expansion and somatic hypermutation of Ig variable heavy chain genes in the synovial membrane of patients with osteoarthritis [J]. J Immunol, 2007,178(1):557-565.
[16] Clemmensen I, Petersen LC, Kluft C. Purificati
上一页 [1] [2] [3] [4] [5] [6] 下一页