I, WILANDER E, DELIUS H, et al. Huamn papillomavirus 16 E6 variants are more prevalent in invasive cervical carcinoma than the prototype [J]. Cancer Res,1998, 58:829833.
[40]XI LF, KOUTSKY LA, GALLOWAY DA, et al. Genomic variation of human papillomavirus type 16 and risk for high grade cervical intraepithelial neoplasia [J]. J Natl Cancer Inst, 1997, 89:796802.
[41]LUXTON J, MANT C, GREENWOOD B, et al. HPV16 E6 oncogene variants in woman with cervical intraepithelial neoplasia [J]. J Med Virol, 2000, 60:337341.
[42]FUJINAGA Y, OKAZAWA K, NISHIHIKAWA Y, et al. Sequence variation of human papillomavirus type 16 E7 in preinvasive cervical neoplasias [J]. Virus Genes, 1994, 9:8592.
[43]WANK R, THOMSSEN C. High risk of squamous cell carcinoma of the cervix for woman with HLADQW3 [J]. Nature, 1991, 352:723725.
[44]STOREY A, THOMAS M, KALTIA A, et al. Role of p53 polymorphism in the development of human papillomavirusassociated cancer [J]. Nature, 1998, 393:229234.
[45]HELLAND A, KARLSEN F, DUE E U, et al. Mutations in the TP53 gene and protein expression of p53, MDM2 and p21/WAF1 in primary cervical carcinomas with no or low human papillomavirus load [J]. Br J cancer, 1998, 78:6972.
[46]WOODWORTH C D, LICHTI U, SIMPSON S, et al.Luekoregulin and Gamainterferon inhibit human papilloma virus type 16 gene transcription in human papillomavirusimmortalized human cervical cells [J]. Cancer Res, 1992, 52:456463.
[47]BRAUN L, DURST M, MIKUMO R, et al. Differential response of nontumorigenic human papillomavirus type 16 positive epithelial cells to transforming growth factor beta 1 [J]. Cancer Res, 1990, 50:73247332.
[48]IGLESIAS M, PLOWMAN G D, WOODWORTH C D. Interleukin6 and interleukin6 soluble receptor regulate proliferation of normal, human papillomavirusimmortalized, and carcinomaderived cervical cells in vitro [J]. Am J Pathol,1995,146:944953.
[49]TARTOUR E, FOSSIEZ
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